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Mitochondrial metabolic dysfunction and non-alcoholic fatty liver disease: new insights from pathogenic mechanisms to clinically targeted therapy | Journal of Translational Medicine | Springer Nature Link

Indeed, the only cell-active small molecules that have been shown to inhibit GPX4 directly act by covalently binding the selenocysteine residue via a reactive α-chloroacetamide group 4,5

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[DOI] [PubMed] [Google Scholar] 168.Balion C., Griffith L., Strifler L., Henderson M., Patterson C., Heckman G., Llewellyn D., Raina P